{"id":2075,"date":"2018-09-28T00:42:45","date_gmt":"2018-09-28T00:42:45","guid":{"rendered":"http:\/\/noti-america.com\/site\/ecuador\/?p=2075"},"modified":"2018-09-28T00:42:45","modified_gmt":"2018-09-28T00:42:45","slug":"una-molecula-detiene-y-revierte-la-neurodegeneracion-que-provoca-el-parkinson","status":"publish","type":"post","link":"https:\/\/noti-america.com\/site\/ecuador\/2018\/09\/28\/una-molecula-detiene-y-revierte-la-neurodegeneracion-que-provoca-el-parkinson\/","title":{"rendered":"Una mol\u00e9cula detiene y revierte la neurodegeneraci\u00f3n que provoca el p\u00e1rkinson"},"content":{"rendered":"

REDACCI\u00d3N NOTI-AM\u00c9RICA (ECUADOR)<\/p>\n

\n

La enfermedad de Parkinson es la segunda patolog\u00eda neurodegenerativa con m\u00e1s prevalencia, despu\u00e9s del alzh\u00e9imer, y es incurable. Se caracteriza por la acumulaci\u00f3n de dep\u00f3sitos de prote\u00ednas en las neuronas dopamin\u00e9rgicas (las que producen dopamina) en forma de fibras amiloides.<\/p>\n

Se trata de agregados formados principalmente por la prote\u00edna alfa-sinucle\u00edna. Esta agregaci\u00f3n se produce de manera muy compleja, y esto hace que sea complicado identificar mol\u00e9culas que puedan prevenir o revertir este proceso y la neurodegeneraci\u00f3n que conlleva.<\/p>\n

Una colaboraci\u00f3n cient\u00edfica liderada por investigadores del Instituto de Biotecnolog\u00eda y de Biomedicina (IBB) de la Universidad Aut\u00f3noma de Barcelona \u200b\u200bha identificado una mol\u00e9cula que detiene y revierte la neurodegeneraci\u00f3n.<\/p>\n

Tras analizar m\u00e1s de 14.000 mol\u00e9culas, han hallado una, a la que han llamado SynuClean-D, que inhibe la agregaci\u00f3n de la prote\u00edna alfa-sinucle\u00edna y rompe las fibras amiloides ya formadas, evitando que se inicie el proceso que desencadena el p\u00e1rkinson.<\/p>\n

\n

\u00abSynuClean-D inhibe la agregaci\u00f3n de la prote\u00edna alfa-sinucle\u00edna y rompe las fibras amiloides ya formadas, evitando que se inicie el p\u00e1rkinson\u00bb<\/em><\/strong><\/p><\/blockquote>\n<\/div>\n

Mediante experimentos realizados con el gusano\u00a0Caenorhabdiis elegans<\/em>, uno de los modelos animales m\u00e1s utilizados para el estudio de enfermedades neurodegenerativas, los investigadores han podido comprobar que, una vez administrada a trav\u00e9s de la comida, la mol\u00e9cula es capaz de reducir notablemente la agregaci\u00f3n de alfa-sinucle\u00edna, impidiendo la propagaci\u00f3n de los agregados t\u00f3xicos y, por tanto, evitando la degeneraci\u00f3n de las neuronas dopamin\u00e9rgicas.<\/p>\n

\u00abTodo parece indicar que la mol\u00e9cula que hemos identificado, la SynuClean-D, puede tener aplicaciones terap\u00e9uticas para tratar enfermedades neurodegenerativas como el p\u00e1rkinson en un futuro\u00bb, apunta Salvador Ventura, coordinador de la investigaci\u00f3n y experto de la UAB.<\/p>\n

Protecci\u00f3n contra la degeneraci\u00f3n neuronal<\/strong><\/p>\n

Para identificar la SynuClean-D, los cient\u00edficos desarrollaron una metodolog\u00eda capaz de hallar inhibidores de la agregaci\u00f3n de la alfa-sinucle\u00edna entre miles de mol\u00e9culas. Una vez identificada, se llev\u00f3 a cabo la caracterizaci\u00f3n biof\u00edsica\u00a0in vitro<\/em>\u00a0de su actividad inhibidora y una prueba de su comportamiento con cultivos de c\u00e9lulas neuronales humanas, antes de testear su comportamiento en modelos animales de la enfermedad (el gusano\u00a0Caenorhabdiis elegans<\/em>).<\/p>\n

Estos animales expresan la alfa-sinucle\u00edna en el m\u00fasculo o en las neuronas dopamin\u00e9rgicas. Los experimentos mostraron c\u00f3mo la administraci\u00f3n del inhibidor identificado reduc\u00eda la agregaci\u00f3n de la prote\u00edna, mejoraba la movilidad del animal y lo proteg\u00eda contra la degeneraci\u00f3n neuronal.<\/p>\n

\n

Referencia bibliogr\u00e1fica:<\/strong><\/p>\n

J.Pujols et al.\u00a0A novel small molecule inhibits \u03b1-synuclein aggregation, disrupts amyloid fibrils and prevents degeneration of dopaminergic neurons,\u00a0PNAS<\/em>. DOI: 10.1073\/pnas.1804198115<\/p>\n

La investigaci\u00f3n ha sido financiada por la Marat\u00f3n de TV3.<\/p>\n<\/div>\n<\/div>\n

Fuente: UAB y SINC<\/div>\n","protected":false},"excerpt":{"rendered":"

REDACCI\u00d3N NOTI-AM\u00c9RICA (ECUADOR) La enfermedad de Parkinson es la segunda patolog\u00eda neurodegenerativa con m\u00e1s prevalencia, despu\u00e9s del alzh\u00e9imer, y es incurable. Se caracteriza por la acumulaci\u00f3n de dep\u00f3sitos de prote\u00ednas en las neuronas dopamin\u00e9rgicas...<\/p>\n","protected":false},"author":17,"featured_media":2076,"comment_status":"open","ping_status":"open","sticky":false,"template":"","format":"standard","meta":{"_acf_changed":false,"om_disable_all_campaigns":false,"rop_custom_images_group":[],"rop_custom_messages_group":[],"rop_publish_now":"initial","rop_publish_now_accounts":{"twitter_242054905_242054905":""},"rop_publish_now_history":[],"rop_publish_now_status":"pending","_uf_show_specific_survey":0,"_uf_disable_surveys":false,"footnotes":""},"categories":[35],"tags":[],"class_list":["post-2075","post","type-post","status-publish","format-standard","has-post-thumbnail","hentry","category-salud"],"acf":[],"aioseo_notices":[],"yoast_head":"\nUna mol\u00e9cula detiene y revierte la neurodegeneraci\u00f3n que provoca el p\u00e1rkinson - Ecuador | Noti-America.com<\/title>\n<meta name=\"robots\" content=\"index, follow, max-snippet:-1, max-image-preview:large, max-video-preview:-1\" \/>\n<link rel=\"canonical\" href=\"https:\/\/noti-america.com\/site\/ecuador\/2018\/09\/28\/una-molecula-detiene-y-revierte-la-neurodegeneracion-que-provoca-el-parkinson\/\" \/>\n<meta property=\"og:locale\" content=\"es_ES\" \/>\n<meta property=\"og:type\" content=\"article\" \/>\n<meta property=\"og:title\" content=\"Una mol\u00e9cula detiene y revierte la neurodegeneraci\u00f3n que provoca el p\u00e1rkinson\" \/>\n<meta property=\"og:description\" content=\"REDACCI\u00d3N NOTI-AM\u00c9RICA (ECUADOR) La enfermedad de Parkinson es la segunda patolog\u00eda neurodegenerativa con m\u00e1s prevalencia, despu\u00e9s del alzh\u00e9imer, y es incurable. 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